Research output per year
Research output per year
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Dive into the research topics where Kyle J Hewitt is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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Projects
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Mechanisms of a GATA Factor-Dependent Hematopoietic Signaling Pathway
Project: Research project
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GATA Factor Mechanisms in Erythroid Regeneration
Project: Research project
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Mechanisms of a GATA Factor-Dependent Hematopoietic Signaling Pathway
Project: Research project
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Hematopoietic Signaling Pathway Mechanism in a GATA Factor-Dependent Network
Project: Research project
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Pancreatic cancer variant analysis of the All of Us cohort
Buckley-McKeown, S. M., Ghosal, G., Hewitt, K. J., Kelly, D. L. & Woods, N. T.
Project: Research project
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Functional requirements for a Samd14-capping protein complex in stress erythropoiesis
Ray, S., Chee, L., Zhou, Y., Schaefer, M. A., Naldrett, M. J., Alvarez, S., Woods, N. T. & Hewitt, K. J., Jun 2022, In: eLife. 11, e76497.Research output: Contribution to journal › Article › peer-review
Open Access -
Sterile α-motif domain requirement for cellular signaling and survival
Ray, S., Chee, L., Matson, D. R., Palermo, N. Y., Bresnick, E. H. & Hewitt, K. J., May 15 2020, In: Journal of Biological Chemistry. 295, 20, p. 7113-7125 13 p.Research output: Contribution to journal › Article › peer-review
Open Access1 Scopus citations -
Dissecting regulatory mechanisms using mouse fetal liver-derived erythroid cells
McIver, S. C., Hewitt, K. J., Gao, X., Mehta, C., Zhang, J. & Bresnick, E. H., 2018, Methods in Molecular Biology. Humana Press Inc., p. 67-89 23 p. (Methods in Molecular Biology; vol. 1698).Research output: Chapter in Book/Report/Conference proceeding › Chapter
7 Scopus citations -
Human leukemia mutations corrupt but do not abrogate GATA-2 function
Katsumura, K. R., Mehta, C., Hewitt, K. J., Soukup, A. A., De Andrade, I. F., Ranheim, E. A., Johnson, K. D. & Bresnick, E. H., Oct 23 2018, In: Proceedings of the National Academy of Sciences of the United States of America. 115, 43, p. E10109-E10118Research output: Contribution to journal › Article › peer-review
24 Scopus citations -
Increased transport of acetyl-CoA into the endoplasmic reticulum causes a progeria-like phenotype
Peng, Y., Shapiro, S. L., Banduseela, V. C., Dieterich, I. A., Hewitt, K. J., Bresnick, E. H., Kong, G., Zhang, J., Schueler, K. L., Keller, M. P., Attie, A. D., Hacker, T. A., Sullivan, R., Kielar-Grevstad, E., Arriola Apelo, S. I., Lamming, D. W., Anderson, R. M. & Puglielli, L., Oct 2018, In: Aging cell. 17, 5, e12820.Research output: Contribution to journal › Article › peer-review
Open Access29 Scopus citations