Abstract
The intracellular bacterium Chlamydia pneumoniae is present in atherosclerotic plaques and may contribute to progression and vulnerability of lesions. Results from animal models support a causal role for C. pneumoniae in atherosclerosis and virulence attributes ascribed to this organism are consistent with this view. Results from recent antibiotic treatment trials have not proven effective, but additional work is needed to define reasons for these failures.
Original language | English (US) |
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Pages (from-to) | 426-429 |
Number of pages | 4 |
Journal | International Congress Series |
Volume | 1262 |
Issue number | C |
DOIs | |
State | Published - May 1 2004 |
Externally published | Yes |
Keywords
- Atherosclerosis
- Chlamydia pneumoniae
- Chronic infection
- GroEL
- Pathogenesis
ASJC Scopus subject areas
- Medicine(all)